Publikasjoner
NIBIOs ansatte publiserer flere hundre vitenskapelige artikler og forskningsrapporter hvert år. Her finner du referanser og lenker til publikasjoner og andre forsknings- og formidlingsaktiviteter. Samlingen oppdateres løpende med både nytt og historisk materiale. For mer informasjon om NIBIOs publikasjoner, besøk NIBIOs bibliotek.
2004
Forfattere
H Schmidt A. Adler Arne Holst-Jensen Sonja Klemsdal C.M. Kullnig-Gradinger Antonio Logrieco C.P. Kibecek Robert L. Mach Rudi F. Vogel Helga I. Nirenberg Ulf Thrane Mona Torp Tapani Yli-Mattila Ludwig NiessenSammendrag
Det er ikke registrert sammendrag
Forfattere
Peder Gjerdrum Olav HøibøSammendrag
Det er ikke registrert sammendrag
Forfattere
Alhaji Jeng Trond Knapp Haraldsen Nils Vagstad Arne Grønlund Steinar TveitnesSammendrag
Nitrogen, fertilizer, meal
Forfattere
Rolf Arnt Olsen Grete RasmussenSammendrag
Det er ikke registrert sammendrag
Sammendrag
The anatomical defense responses in stems of Norway spruce (Picea abies) clones of different resistance to pathogenic fungi were characterized over time and distance from small mechanical wounds or wounds inoculated with the root rot fungus Heterobasidion annosum. Common responses for both treatments included division of ray parenchyma and other cells in the cambial zone, accumulation of phenolic inclusions in ray parenchyma cells, activation of phloem parenchyma (PP) cells, and formation of traumatic resin ducts (TDs) in the xylem. TD formation occurred synchronously from a tangential layer of cells, or symplasmic domain, within the zone of xylem mother cells. TD induction is triggered by a signal, which propagates a developmental wave in the axial direction at about 2.5cm per day. TDs are formed at least 30cm above single inoculations within 16–36days after inoculation. The size and number of TDs is attenuated further away from the inoculation site, indicating a dose-dependent activity leading to TD development. Compared to sterile wounding, fungal inoculation gave rise to more and larger TDs in all clones, and multiple rows of TDs in weak clones. Fungal inoculation also induced the formation of more new PP cells, increasing the number of PP cells in the phloem in the year of inoculation up to 100%. TD and PP cell formation was greater in susceptible compared to resistant clones and after fungal versus sterile inoculation. Potential mechanisms responsible for this variable response are discussed.
Forfattere
Live Semb Vestgarden Petter Nilsen Gunnar AbrahamsenSammendrag
Det er ikke registrert sammendrag
Forfattere
Halvor B. Gjærum Kåre Arnstein Lye Halvor SolheimSammendrag
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Sammendrag
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Forfattere
Nina Elisabeth Nagy Carl Gunnar Fossdal Paal Krokene Trygve Krekling Anders Lönneberg Anders Lønneborg Halvor SolheimSammendrag
Polyphenolic parenchyma cells (PP cells) in Norway spruce (Picea abies (L.) Karst.) stem phloem play important roles in constitutive and inducible defenses. To determine whether anatomical and molecular changes in PP cells are correlated with tree resistance, we infected two Norway spruce clones with the pathogenic fungus Ceratocystis polonica (Siem.) C. Moreau. The fungus induced significantly different lesion lengths in the two clones, indicating that one clone was more resistant to the fungus (short lesions) than the other (long lesions). After infection, the cross-sectional area of PP cells and their vacuolar polyphenol bodies increased in the three most recent annual rings of PP cells in both clones. The more resistant clone had larger PP cells with denser polyphenol bodies than the less resistant clone, whereas the less resistant clone accumulated relatively more polyphenols after infection. Compared with the less resistant clone, the more resistant clone contained higher starch concentrations before infection that were reduced more quickly after infection before returning to original values. Low transcript levels of chalcone synthase were detected in uninfected tissues of both clones, but the levels increased dramatically after infection. Transcript levels were higher and peaked 6 days earlier in the more resistant clone than in the less resistant clone. The activity of at least one highly basic peroxidase isoform was greatly enhanced after infection, and this increase occurred earlier in the more resistant clone.
Forfattere
Steen Koekebakker Gudbrand LienSammendrag
Det er ikke registrert sammendrag